A recent study may tip the scales regarding the method and treatment to stop a disease Alzheimer’s Which puzzled scientists for decades, and who believed that the only disease is related only to the brain.
A group of scientific trials conducted by British researchers revealed that the intestine represents an alternative target that may be easier to influence with drugs or diet changes to stop dementia.
A conference in Brighton, Britain, on Wednesday, featured a series of experiments linking the gut to the development of Alzheimer’s disease, according to the British newspaper, “Daily Mail”.
gut microbiome
In addition, one of the trials presented at the conference revealed how the gut microbiomes differ significantly in patients with Alzheimer’s from those without the disorder.
Alzheimer’s (Shutterstock)
Another trial found that rodents given “faecal” transplants directly from Alzheimer’s patients performed worse on memory tests.
Inflammation levels
In parallel, a third experiment found that brain stem cells treated with blood from patients with the disorder were less able to build new neurons.
Patients’ gut bacteria affect levels of inflammation in the body, which then affects the brain via the blood supply. Also, inflammation is a major factor in the development of Alzheimer’s disease.
Dr Idina Silajic, a neuroscientist from King’s College London who took part in analyzing samples from Alzheimer’s patients, said most people are surprised that gut bacteria can have any effect on their brain health.
An Alzheimer’s patient (Archyde.com)
Evidence for this is mounting, she added, noting that scientists are building their understanding of how this happens.
Alzheimer’s discovery
It is reported that Alzheimer’s was discovered by Dr. Alius Alzheimer in 1906, who observed changes in the brain tissue of a woman who had died of an unusual mental illness.
Doctors usually diagnose Alzheimer’s disease when they find a mixture of amyloid plaques and neurofibrillary tangles, along with the dimensions.
The abnormal buildup is known to cause Alzheimer’s disease, which includes two types of proteins: one called amyloid, whose deposits form plaques around brain cells, and another called tau, which forms tangles inside brain cells.
